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Internuclear ophthalmoplegia or ophthalmoparesis (INO) is an ocular movement disorder that presents as an inability to perform conjugate lateral gaze and ophthalmoplegia due to damage to the interneuron between two nuclei of cranial nerves (CN) VI and CN III (internuclear). [1] This interneuron is called the medial longitudinal fasciculus (MLF).

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Internuclear ophthalmoparesis (INO), also commonly referred to as internuclear ophthalmoplegia, is a specific gaze abnormality characterized by impaired horizontal eye movements with weak and slow adduction of the affected eye, and abduction nystagmus of the contralateral eye. It is one of the most localizing brainstem syndromes, resulting from.

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An internuclear ophthalmoplegia (INO) follows disruption of the medial longitudinal fasciculus (MLF). In a right INO, there is impairment of adduction of the right eye. The defect may be partial or complete, and it is present whether the eye movements are volitional or reflex. If convergence is preserved, it can be assumed that the MLF lesion.

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